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  1. (2012) suggested that loss of function of proteins that have a dual role in centrosome and DNA repair signaling could cause a disturbance of cell-cycle checkpoint controls, which is detrimental to progenitor cell survival both during embryogenesis and later during tissue maintenance. (2012) found that knockdown of Cep164 in zebrafish resulted in a ciliopathy with ventral body axis curvature, cell death, abnormal heart looping, pronephric tubule cysts, hydrocephalus, and retinal dysplasia.